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Rickets

Rickets is the softening and weakening of bones in children, usually because of an extreme and prolonged vitamin D deficiency.

Vitamin D promotes the absorption of calcium and phosphorus from the gastrointestinal tract. A deficiency of vitamin D makes it difficult to maintain proper calcium and phosphorus levels in bones, which can cause rickets.

If a vitamin D or calcium deficiency causes rickets, adding vitamin D or calcium to the diet generally corrects any resulting bone problems for your child. Rickets due to a genetic condition may require additional medications or other treatment. Some skeletal deformities caused by rickets may need corrective surgery.

Symptoms Causes Risk factors Complications Prevention

Signs and symptoms of rickets may include:

  • Delayed growth
  • Pain in the spine, pelvis and legs
  • Muscle weakness

Because rickets softens the growth plates at the ends of a child's bones, it can cause skeletal deformities such as:

  • Bowed legs
  • Thickened wrists and ankles
  • Breastbone projection

When to see a doctor

Talk to your doctor if your child develops bone pain, muscle weakness or obvious skeletal deformities.

Your body needs vitamin D to absorb calcium and phosphorus from food. Rickets can occur if your child's body doesn't get enough vitamin D or if his or her body has problems using vitamin D properly.

Lack of vitamin D

You receive vitamin D from two sources:

  • Sunlight. Your skin produces vitamin D when it's exposed to sunlight. But children in developed countries now tend to spend less time outdoors. They're also more likely to use sunscreen, which blocks the rays that trigger the skin's production of vitamin D.
  • Food. Fish oils, fatty fish and egg yolks contain vitamin D. Vitamin D also has been added to some foods, such as milk, cereal and some fruit juices. Children who don't eat enough of these fortified foods can develop a vitamin D deficiency.

Problems with absorption

Some children are born with or develop medical conditions that affect the way their bodies absorb vitamin D. Some examples include:

  • Celiac disease
  • Inflammatory bowel disease
  • Cystic fibrosis
  • Kidney problems

Factors that may increase a child's risk of rickets include:

  • Age. Children 3 to 36 months old are most at risk of rickets because their skeletons are growing so rapidly.
  • Dark skin. Dark skin doesn't react as strongly to sunshine as does lighter colored skin, so it produces less vitamin D.
  • Northern latitudes. Children who live in geographical locations where there is less sunshine are at higher risk of rickets.
  • Premature birth. Babies born before their due dates are more likely to develop rickets.
  • Anti-seizure medications. Certain types of anti-seizure medications appear to interfere with the body's ability to use vitamin D.
  • Exclusive breast-feeding. Breast milk doesn't contain enough vitamin D to prevent rickets. Babies who are exclusively breast-fed should receive vitamin D drops.

If left untreated, rickets may lead to:

  • Failure to grow
  • Abnormally curved spine
  • Skeletal deformities
  • Dental defects
  • Seizures

Most adolescents and adults receive much of their necessary vitamin D from exposure to sunlight. Infants and young children, however, need to avoid direct sun entirely or be especially careful by always wearing sunscreen.

Make sure your child is eating foods that contain vitamin D naturally — fatty fish, fish oil and egg yolks — or that have been fortified with vitamin D, such as:

  • Infant formula
  • Cereals
  • Milk
  • Orange juice

Because human milk contains only a small amount of vitamin D, all breast-fed infants should receive 400 international units (IU) of oral vitamin D daily.

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